克劳斯教授(Prof. Ronald M. Krauss)是美国儿科学教授、医学研究员。他研究遗传、饮食和激素对血浆脂蛋白和冠心病风险的影响。他和他的同事也是首批确定LDL亚型对人体健康的影响具有显著差异的科学家。
克劳斯是加州大学伯克利分校儿科和医学教授，加州大学伯克利分校营养科学教授。他在哈佛医学院获得医学学位，并在美国国立卫生研究院接受博士后培训。他获得了内科、内分泌和新陈代谢的委员会认证，也是美国心脏协会的会员和美国临床研究协会的成员。
克劳斯对当前的饱和脂肪指南提出异议。克劳斯认为，“与碳水化合物产生的量相比，我们在饮食中消耗的饱和脂肪量对我们体内的饱和脂肪的影响相对较小。”在 2016 年接受马克·海曼医生的采访中，克劳斯教授表示，他的研究并未发现饱和脂肪摄入与心脏病风险增加之间存在联系，而碳水化合物会增加风险。
Ronald M. Krauss is an American Professor of Pediatrics, medical researcher and low-carbohydrate diet advocate. He studies genetic, dietary, and hormonal effects on plasma lipoproteins and coronary disease risk.
Krauss is Professor of Pediatrics and Medicine, UCSF, and Adjunct Professor of Nutritional Sciences, University of California at Berkeley. He received his medical degree at Harvard Medical School and received postdoctoral training at the National Institutes of Health. He is board certified in internal medicine, endocrinology and metabolism.
He has disclosed that he received grants from the National Cattlemen’s Beef Association and National Dairy Council. He is also on the scientific advisory board of Virta Health, a company that provides ketogenic diet counselling for people with diabetes.
Krauss is a Fellow of the American Heart Association and member of the American Society for Clinical Investigation.
Krauss disputes the current saturated fat guidelines. Krauss has argued that “the amount of saturated fat that we consume in our diet has a relatively small effect on the saturated fat in our body compared with the amount that is produced from carbohydrates.” In a 2016 interview with Mark Hyman, Krauss stated that his research has not found a connection between saturated fat intake and increased heart disease risk and that it is carbohydrates that increase the risk.
信息来源：https://en.wikipedia.org/wiki/Ronald_Krauss
加州大学旧金山分校、加州大学伯克利分校官方介绍
克劳斯教授的研究项目侧重于血浆脂蛋白代谢和影响冠状动脉疾病风险的相关特征。他的实验室开发和应用的方法导致发现了一种常见的受遗传影响的致动脉粥样硬化脂蛋白表型，该表型是肥胖、2 型糖尿病和代谢综合征患者心血管疾病风险的基础。
目前的一个主要研究重点是确定对他汀类药物治疗的表型和临床反应的广泛个体间变异性的遗传决定因素。正在检查多个他汀类药物治疗的人群样本，以测试来自候选基因和全基因组搜索与他汀类药物功效标志物以及肌肉毒性的 SNP 关联的结果的可重复性和普遍性。他正在研究适当的人类细胞和动物模型中评估遗传发现的功能意义，目的是将基因型与心血管疾病风险的临床相关决定因素联系起来。
Dr. Krauss’ research program focuses on plasma lipoprotein metabolism and related traits that influence risk for coronary artery disease. His laboratory developed and applied methodology that led to the discovery of a common genetically-influenced atherogenic lipoprotein phenotype that underlies cardiovascular disease risk in patients with obesity, type 2 diabetes, and metabolic syndrome. A major current research emphasis has been the identification of genetic determinants of the wide range of inter-individual variability in phenotypic and clinical response to statin treatment. Multiple statin-treated population samples are being examined to test the reproducibility and generalizability of findings derived from both candidate gene and genome-wide searches for SNP associations with markers of statin efficacy as well as muscle toxicity. Functional significance of genetic findings is being evaluated in appropriate human cellular and animal models, with the goal of connecting genotypes to clinically relevant determinants of cardiovascular disease risk.
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